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Please contact support. Create a new Playlist. Variations to the sex gene karyotype could include rare disorders such as XX males often due to translocation of the SRY gene to the X chromosome or XY gonadal dysgenesis in people who are externally female due to mutations in the SRY gene.
The XY system contrasts in several ways with the ZW sex-determination system found in birdssome insects, many reptilesand various other animals, in which the heterogametic sex is female.
It had been thought for several decades that in all snakes sex was determined by the ZW system, but there had been observations of unexpected effects in the genetics of species in the families Boidae and Pythonidae ; for example, parthenogenic reproduction produced only females rather than males, which is the opposite of what is to be expected in the ZW system.
In the early years of the 21st century such observations prompted research that demonstrated that all pythons and boas so far investigated definitely have the XY system of sex determination. A temperature-dependent sex determination system is found in some reptiles. All animals have a set of DNA coding for genes present on chromosomes. In humans, most mammals, and some other species, two of the chromosomescalled the X chromosome and Y chromosomecode for sex.
In these species, one or more genes are present on their Y chromosome that determine maleness.
In this process, an X chromosome and a Y chromosome act to determine the sex of offspring, often due to genes located on the Y chromosome that code for maleness. Offspring have two sex chromosomes: an offspring with two X chromosomes will develop female characteristics, and an offspring with an X and a Y chromosome will develop male characteristics.
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In humans, half of spermatozoons carry X chromosome and the other half Y chromosome. Presence of this gene starts off the process of virilization. This and other factors result in the sex differences in humans. The inactivated X chromosome remains within a cell as a Barr body. Humans, as well as some other organisms, can have a rare chromosomal arrangement that is contrary to their phenotypic sex; for example, XX males or XY gonadal dysgenesis see androgen insensitivity syndrome.
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Additionally, an abnormal number of sex chromosomes aneuploidy may be present, such as Turner's syndromein which a single X chromosome is present, and Klinefelter's syndromein which two X chromosomes and a Y chromosome are present, XYY syndrome and XXYY syndrome. In most mammals, sex is determined by presence of the Y chromosome. SRY is a sex-determining gene on the Y chromosome in the therians placental mammals and marsupials.
Not all male-specific genes are located on the Y chromosome.
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Platypusa monotremeuse five pairs of different XY chromosomes with six groups of male-linked genes, AMH being the master switch. Birds and many insects have a similar system of sex determination ZW sex-determination systemin which it is the females that are heterogametic ZWwhile males are homogametic ZZ. Many insects of the order Hymenoptera instead have a system the haplo-diploid sex-determination systemwhere the males are haploid individuals which have just one chromosome of each typewhile the females are diploid with chromosomes appearing in pairs.
Some other insects have the X0 sex-determination systemwhere just one chromosome type appears in pairs for the female but alone in the males, while all other chromosomes appear in pairs in both sexes.
It has long been believed that the female form was the default template for the mammalian fetuses of both sexes. After the discovery of the testis-determining gene SRYmany scientists shifted to the theory that the genetic mechanism that causes a fetus to develop into a male form was initiated by the SRY gene, which was thought to be responsible for the production of testosterone and its overall effects on body and brain development.
This perspective still shares the classical way of thinking; that in order to produce two sexes, nature has developed a default female pathway and an active pathway by which male genes would initiate the process of determining a male sex, as something that is developed in addition to and based on the default female form. However, In an interview for the Rediscovering Biology website,  researcher Eric Vilain described how the paradigm changed since the discovery of the SRY gene:.
For a long time we thought that SRY would activate a cascade of male genes. It turns out that the sex determination pathway is probably more complicated and SRY may in fact inhibit some anti-male genes.
The idea is instead of having a simplistic mechanism by which you have pro-male genes going all the way to make a male, in fact there is a solid balance between pro-male genes and anti-male genes and if there is a little too much of anti-male genes, there may be a female born and if there is a little too much of pro-male genes then there will be a male born. We [are] entering this new era in molecular biology of sex determination where it's a more subtle dosage of genes, some pro-males, some pro-females, some anti-males, some anti-females that all interplay with each other rather than a simple linear pathway of genes going one after the other, which makes it very fascinating but very complicated to study.
In mammals, including humans, the SRY gene is responsible with triggering the development of non-differentiated gonads into testes, rather than ovaries. However, there are cases in which testes can develop in the absence of an SRY gene see sex reversal.
In these cases, the SOX9 gene, involved in the development of testes, can induce their development without the aid of SRY. Even so, the absence of the SRY gene or the silencing of the SOX9 gene are not enough to trigger sexual differentiation of a fetus in the female direction.